Influence of Nutrition on Premetabolic Syndrome and Vascular Variability Syndrome

The metabolic syndrome (MS) is a constellation of abnormalities including central obesity; glucose intolerance, and type 2 diabetes; hypertension; and a dyslipidemia characterized by increased serum triglycerides, decreased high-density lipoprotein (HDL) cholesterol, and increased small, dense low-density lipoprotein (LDL) particles [1-6]. Presence of biophysical-semeiotic constitution, and related inherited real risks evolving slowly towards relative disorders, may be defined as premetabolic syndrome. The metabolic syndrome affects more than 27% of adults in the United States [6] and increases the risk of cardiovascular disease 2to 3-fold, but exclusively in individuals with biophysical-semeiotic constitution-dependent, inherited, coronary real risk, which has been described in the former paper on this website, thus not in all patients with MS [7-12]. More precisely speaking, all components of metabolic syndrome may occur exclusively in subjects with congenital acidosic enzyme metabolic histangiopathy (CAEMH), as well as with either "some" or all CAEMH-dependent, biophysical-semeiotic constitutions [4, 5, 7, 9-12]. As a consequence, not all patients with metabolic syndrome are equal. Biophysical-semiotic constitution may be characterised with abnormal vasomotion in the microcirculation.